Altered ion channel trafficking as the basis of autoimmune and neuropathic pain.

My lab focuses on studying rare autoimmune clinical conditions to discover novel proteins involved in the transition to chronic pain. To achieve this goal, we use a variety of in vitro and in vivo approaches, from biochemistry and CRISPR to electrophysiology and pain behavior. My goal is to better understand the synaptic dysregulations underlying the increased spinal neurotransmission in chronic pain conditions.